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Studies have shown that the stimulation of D4R on human T cells promotes quiescence (152), and overexpression of D4R in SLE patients may act as a compensatory mechanism to inhibit uncontrolled T cell proliferation, an important link in the pathogenesis of SLE

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Here, we report additional EOT data and preliminary off-treatment responses

It aids in coping with stress in various ways e Nuclear Overhauser Effect (NOEs) reporting on the motion of individual N-H bond vectors and providing information about the rigidity of the protein backbone, (iii) R 1 and R 2 relaxation rates reporting on overall tumbling and internal motions