Encouraging data support the use of nutraceuticals for female reproductive disorders
Effects of oxidative stress on viral infections: an overview
actioneaza si la nivel cerebral datorita capacitatii de a traversa bariera hemato-encefalica: glutationul redus este principalul transportator al gruparilor SH (sulfhidril) libere care au o mare capacitate de a neutraliza radicalii liberi prin cedarea unei perechi de electroni reducerea actiunii enzimelor proinflamatoare asupra celulelor cu membrane bogate in acizi grasi polinesaturati (de ex.: celulele imunitare, spermatozoizii, hepatocitele, neuronii etc.) participa la faza I si II de detoxifiere hepatica: reducerea absorbtiei si favorizarea eliminarii din organism a substantelor nocive endogene (hormoni in exces, enzime proinflamatoare, melanina s.a.) si a xenobioticelor (metale grele, fum de tigara, alcool, pesticide, fungicide, erbicide, coloranti si aditivi alimentari toxici [de ex.: formaldehida], droguri, microorganisme nocive, detergenti, grasimi vegetale hidrogenate, nitrozamine, metilglioxal s.a.) stimularea secretiei de gama-interferon, interleukina IL-2, limfocite T citotoxice, NK, activarea macrofagelor si granulocitelor cu rol in reglarea diviziunii celulare normale si reducerea actiunii agentilor agresori cu potential de modificare a ADN-ului si ARN-ului celular mentinerea nivelului de glutation in stari cu deficit de sinteza cauzat de microorganisme nocive, droguri, alcool, anticonceptionale, amoniac, metale grele, pesticide, hormoni, consum redus de proteine, diviziune celulara anormala, chimio si radioterapie, interventii chirurgicale, arsuri, inaintarea in varsta etc
Extended cycles may produce greater cumulative benefits, though research specifically supporting this approach remains limited
Omega-7s are unsaturated fatty acids that have shown promising clinical results for whole-body health and can support collagen production
7 It remains elusive whether common key molecular mediators exist in the pathophysiology of both SZ and BP, and if they connect shared genetic risks with gross phenotypic manifestations at the anatomical and clinical levels
