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Tumor cells have increased iron ion content compared to normal cells, and activation of Nrf2 can HO-1 activation, while excessive activation of HO-1 can cause cellular iron overload, induce cellular ferroptosis and exert cytotoxicity

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however, GSH levels are also influenced by agents or conditions that alter the thiol redox state, that lead to the formation of glutathione S -conjugates or complexes, and/or that disrupt the distribution of GSH among various intracellular organelles

These data emphasize the controversial effects of ROS and careful considerations in Nox inhibitor development aiming to reduce ROS levels

Dendrimers-based drug delivery system: a novel approach in addressing Parkinsons disease

Elucidating the contribution of mitochondrial glutathione to ferroptosis in cardiomyocytes