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METH transit across the BBB damages the nucleus accumbens shell region and prefrontal cortex and causes hyperthermia, neuroinflammation, and brain edema (Zhang et al., 2017)

Collectively, these findings establish a TRPV1-dependent thermogenic program in adipose tissue, providing a mechanistic framework for interpreting human studies in which dietary capsaicin or capsinoids modestly increase energy expenditure, particularly in individuals with metabolically active brown adipose tissue (2023)

New York, NY, USA: Springer International Publishing

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An interactive resource to identify cancer genetic and lineage dependencies targeted by small molecules