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The binding of AMP to the subunit induces allosteric activation of AMPK, whereas ATP can inhibit this activation (58)

The slow onset of therapeutic effect is attributed to the time it takes for these compounds to alter gene expression (see glucocorticoid_pharmacology, which produces: reduced synthesis of inflammatory cytokines reversal of inflammation-induced increases of capillary permeability reduced tissue edema depression of the migration of a host of inflammatory cells into lung tissue (e.g

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Diabetes 49 (11), 19391945

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