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Molecular diagnosis of Menkes disease: genotypephenotype correlation

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[1] At the cellular level, the tendon work is genuinely mechanistic: in cultured rat tendon fibroblasts, BPC-157 accelerated cell outgrowth, improved survival under oxidative stress, and increased migration in a dose-dependent way, apparently through the FAKpaxillin pathway

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These findings are preclinical

As the diversity and abundance of microbes increase from the upper GI to distal sites, the number of iEANs likewise increases