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Mutations in AR receptor, AR overexpression, intracrine production, contorted expression of enzymes and cofactors of AR, autonomous activation of AR signaling by cytokines in the absence of androgen ligands, and the presence of multiple splice variants of AR are the main causative agents responsible for the therapeutic failure [12, 13]

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10.7150/ijbs.76140 59 GaoQ.ShengQ.YangZ.ZhuZ.LiL.XuL.et al (2025)

doi: 10.3390/ani11010136 13 MeyerJDanielsSUGrindlerSTrscher-MuotterJAlaedinMFrahmJet al

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