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[DOI] [PubMed] [Google Scholar] Catani M, ffytche DH

V., Hasegawa, P

The Journal of laboratory and clinical medicine, 133(6), 575582

Mitchell et al.[6] measured the level of GSH depletion in the liver (of mice), and the amount of covalent binding of radiolabeled APAP metabolites (later identified as NAPQI) in the liver after a wide range of doses of APAP

Mechanistically, EGCG co-treatment led to increased apoptosis and DNA damage compared to either agent alone, as evidenced by elevated levels of cleaved caspase-3 and PARP, a decrease in antiapoptotic Bcl-2, and an increase in pro-apoptotic Bad (60)

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