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In the context of AD, the abnormal immune response leads to elevated levels of cytokines such as IL-2, resulting in increased production of ROS and lipid peroxidation (91)

Data suggests that GHK can upregulate certain insulin-related genes while downregulating others, with links to studies proposing a relationship between reduced insulin/IGF-1 signaling and delayed cellular aging

This relationship is reciprocal since thyroid conditions can worsen OxS and increase ROS production, exacerbating oxidative damage