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endometriosis and l'carnitine

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Integrating these emerging yet scattered observations, we propose a therapeutic strategy that exploits metabolic vulnerabilities to exacerbate genome instability

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TBK1-mediated phosphorylation of NDP52 facilitates direct interaction with the FIP200/ULK1 complex thus enabling spatial proximity to ubiquitinated cargo to drive autophagosome biogenesis (97)

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