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For example, high levels of iNOS and NFB expression were shown in the peripheral blood of primary patients with Echinococcus granulosus , while their low expression occurred in the relapsed patients (214)

This structural damage directly impairs the carnitine shuttle and beta-oxidation pathways

[Preventive cytoprotection in social diseases]

At the cellular level, OXPHOS impairment triggers a cascade of pathophysiological events, most notably inducing a hypermetabolic state

Harikumar KB, Nimita CV, Preethi KC, Kuttan R, Shankaranarayana ML, Deshpande J

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