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These data support the notion that exogenous CoQ10 supplementation can suppress lipid peroxidation, protect dopaminergic neurons, and inhibit -synuclein aggregation in PD models (281), highlighting the therapeutic potential of CoQ10-based interventions

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doi: 10.1111/and.13162 59 HussainTKandeelMMetwallyEMurtazaGKalhoroDHYinYet al

Figure 1 Although the mechanisms of epilepsy vary across different mitochondrial diseases, Figure 1 outlines common pathways of cell death associated with epilepsy