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Recent studies have suggested diverse mechanisms as underlying demyelination, including a subset of lesions induced by an interaction between metabolic insult to oligodendrocytes and inflammatory mediators

doi: 10.1021/acsnano.4c02420 107 GoodmanRSJohnsonDBBalkoJM

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Unlike GLP-1 analogs that primarily modulate insulin secretion and appetite through the glucagon-like peptide-1 pathway, cagrilintide offers a complementary pathwaywhich is why its frequently studied in conjunction with semaglutide and tirzepatide

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