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Histamine induces vascular hyperpermeability by increasing blood flow and endothelial barrier disruption in vivo

Mechanistically, it was shown that OA markedly ameliorated mitochondrial injury induced by hypoxia-reoxygenation through the enhancement of the PPAR-BNIP3 signaling pathway

Although such difference in the inhibition behavior of EA toward FXIIIa-mediated dimerization and polymerization of fibrin(ogen) monomers is under investigation, the results indicate that the inhibition activity of EA toward the catalytic activity of human FXIIIa is physiologically relevant

Notably, cerebral dysfunction has been identified as a potential extrahepatic symptom of NAFLD (284)

doi: 10.1038/srep42095 238 EspinozaJALenMACspedesPFGmezRSCanedo-MarroqunGRiquelmeSAet al

Lu, Hormonal and cell density regulation of hepatic gamma-glutamylcysteine synthetase gene expression