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Additionally, post-operative IF prevented microglial activation and the elevation of sphingosine-1-phosphate receptor 1 and inflammatory cytokines in the hippocampus of 2VO rats

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doi: 10.1093/rheumatology/41.12.1395 11 LernerAJeremiasPMatthiasT

5.3.3 Progress in drugs targeting A1 astrocytes The transformation of astrocytes into the neurotoxic A1 phenotype following TBI represents a critical factor exacerbating secondary injury

GCDH loss did not alter 2-OG metabolism, nor 2-HG levels