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[DOI] [PubMed] [Google Scholar] [105].Munoz-Pinedo C, Guio-Carrion A, Goldstein JC, Fitzgerald P, Newmeyer DD, Green DR

This discrepancy of iron chelation worsening neurodegeneration characterised by local iron overload raises a critical question: why does iron chelation block ferroptosis induced by pro-ferroptotic agents such as RSL3 and erastin (where there is no iron elevation in cell culture), yet fail to protect in the context of human neurodegenerative disease where iron is elevated

There are some risks to glutathione supplementation, however, they are rare

Neutral amino acid transporters (NAATs), typically the alanine serine cysteine transporter (ASCT) and the L-type amino acid transporter 2 (LAT2), mediate the cellular uptake of Cys in ordinary cells [46]

Additionally, polymorphisms in GPX1 (e.g., Pro198Leu, rs1050450) have been associated with decreased enzymatic activity and increased OS, potentially exacerbating autoimmune thyroid conditions