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Christian Zidorn, Christian-Albrechts-Universitt zu Kiel, Germany Updates Copyright 2018 Czerwiska and Melzig

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TNF- damages cardiomyocyte mitochondria by inhibiting glycolysis and reducing mitochondrial adenosine triphosphate (ATP) production through promoting oxidative and nitrosative stress, which can result in compromised cardiac systolic function and unfavorable cardiac remodeling [83, 84]

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