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To understand the strong association between DS and AD neuropathology, first it is necessary to consider the trisomy of Chr21, in which the overexpression of selected genes already has been identified to be involved in the development of AD neuropathology in the general population (457) (FIGURE 11)

We further extended this observation in a panel of breast cancer cell lines doing clonogenic assays with 1214 days treatment

At physiological levels, they function as crucial signaling entities that regulate cell growth, differentiation, and survival [3]

Therapeutic advances in drug safety, 4(3), 125133