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When these mutations occur, they grant mutant clones a selective growth advantage, enabling them to evade normal cell-cycle checkpoints and apoptotic signals [80,81,82,83]

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At 150 days post-chemo, 5/9 from the CDP + NMN group had litters, yielding 32 pups, compared to zero litters from the CDDP alone group (Fig

Considering the limited capacity of DCs to oxidize intracellular lipids, these truncated lipids are likely derived from the TME (Ramakrishnan 2014;, Veglia 2017), and lipid peroxidation in tumor cells is a significant source

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and Xu, S