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To maintain a balance between its functional roles and potential for generation of harmful reactive oxygen species, cellular levels of copper are tightly regulated by a network of proteins responsible for its influx, efflux, transport and storage 1,2

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Clin Sci 132:12431252

In RTK-mediated RAS signaling, the mTORC1 pathway is activated via MAPK/ERK and its effector p90RSK through the RTK RAS MAPK/ERK p90RSK cascade, further resulting in the phosphorylation of TSC2

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