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Redox homeostasis is controlled by a variety of cell defense mechanisms, including three different superoxide dismutase (SOD1 in cells, SOD2 in mitochondria, SOD3), glutathione peroxidase (GPX), and signal pathways Nuclear factor erythroid 2-related factor 2 (NRF2)- Kelch-like ECH-associated protein 1(KEAP1) (71)

many other glycosylation-prone targets have been identified that link nutrient availability with growth signaling 105,106,107,108,109,110 and this branch of research deserves more attention 111

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Lett Drug Des Discovery 16(10):11671174 Laun AS et al

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In contrast, cancers with high expression of only one or two enzymes display metabolic flux redirection or environmental dependence, requiring combination strategies targeting transporter bypass pathways or microenvironmental remodeling cascades