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Future studies should prioritize: in vivo evaluation in infected wound models to assess healing efficacy and safety, mechanistic investigations into antimicrobial and antibiofilm pathways, extended release studies beyond 24 h to correlate with chronic wound healing timelines, and formulation optimization for scalability and regulatory compliance

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ZDHHC2 deficiency attenuates pathological progression in mice by inhibiting pro-inflammatory cytokine expression in inflamed skin and leukocyte infiltration in psoriatic lesion areas, and ZDHHC2 deficiency reduces migration of pDC to the skin and other organs, which reduces psoriasis risk (151)