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In calves, Sertoli cells, Se improved cell viability and expression of bloodtestis-barrier proteins (occludin, connexin-43, zonula occluden, E-cadherin) by modulating the expression of mitochondria-related genes, inhibiting the nuclear factor kappa B (NFB) activation of inflammatory cytokines and apoptosis, and increasing the GPX4 activity (160, 161)

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The absence of a listed interaction is not the same as proof of safety, but the pharmacokinetic analysis above explains why no interaction would be expected

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