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Cancer cells acquire a diverse range of metabolic flexibility and plasticity as an adaptation to ever-changing nutritional microenvironments during tumor evolution [13]

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In ApoE / animals subjected to a high-fat diet, Double-NPs significantly accumulated in atherosclerotic plaques, decreasing plaque area, inflammatory infiltration, and instability [171]

[1] The IonescuFrohman work then characterized the important finding that pulsatility is preserved under sustained stimulation

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