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Following acute stress, an increase in adenosine release triggers an adaptive response aimed at restoring homeostasis, mainly by activating A 1 receptors that promote an inhibitory control of neuronal hyperexcitability [54]

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5.4 Potential implications of combined therapies on patient outcomes Although direct clinical evidence supporting combined ferroptosis and cuproptosis targeted therapy remains limited, combined ferroptosis- and cuproptosis-targeted strategies may hold potential to improve patient prognosis across multiple biological and clinical dimensions