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However, contradictory results have been recently reported, in which HSV-1 infection produced SSBs and DSBs in rat embryo cortical neurons and reduced the expression of Ku80, a component involved in NHEJ which is involved in DSB repair (De Chiara et al., CNS Inflammation Induced by HSV-1 Role of Toll-Like Receptors in Brain Inflammation by HSV-1 TLRs are components of the immune system that contribute at providing the host a first line of defense against viral infections (Xagorari and Chlichlia, 2008)