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This epigenetic memory enables adaptive evolution under therapeutic pressure, fostering heterogeneous subpopulations with enhanced drug tolerance, thus ultimately leading to therapeutic failure upon rechallenge [22]

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Although amide bonds are often used to link the various components of PROTACs, it should be noted that these bonds are still susceptible to hydrolytic or enzymatic cleavage, resulting in a loss of potency of the PROTAC 92

One published review article compiled decades of research in BioMed Research International that support its regenerative benefits