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Reports have demonstrated that -KG coordinates the M1/M2 phenotypic balance of alveolar macrophages via immune response modulators (PPAR or mTORC1), thereby impacting sepsis-associated ALI/ARDS (323)

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They then amplified the HAGST-7 gene, and found a variant that differed in amino acid sequence from control larvae, so they named it HAGST-8 [8]

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