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Most ALPS patients are affected by heterozygous germline mutations in the Fas cell surface death receptor gene ( FAS ) via autosomal dominant inheritance (Jackson et al., 2010)

To confirm our observation of low B12-induced FAO impairment in AT, we assessed mitochondria functional integrity in a preadipocyte cell line and primary adipocytes, and the correlation of B12 with FAO genes in mature human scWAT and omWAT

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