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Notably, the therapy induced immunogenic cell death, suggesting the potential involvement of immune system reactivation in the treatment response (304)

Generally, these findings demonstrate that metal-specific metallochaperone, iron transport (e.g., DMT1), and cellular metabolism together regulate the iron physiological state

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It is, however, noteworthy to mention that for a number of glycolytic enzymes, we did not find the reported most critical cysteine residue (i.e., the active-site cysteine or key cysteine for enzymatic function) to be more oxidized in tumors