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In this study, we applied NMR-based metabonomics to obtain an understanding of metabolic dysregulation driven by common KRAS mutations and also studied the effects of single copy mutant KRAS on the cellular metabolic profile

3C, with bar lengths representing the target counts, and the KEGG pathways with higher relevance were Th17 cell differentiation, HIF-1 signaling pathway, IL-17 signaling pathway, PI3K-Akt signaling pathway, NF-kappa B signaling pathway, TNF signaling pathway, Th1 and Th2 cell differentiation, and so on

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In AD and related tauopathies, hyperphosphorylated tau assembles into neurofibrillary tangles that impair proteasome activity and disrupt cellular homeostasis