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whereas, its high level can cause damage to cell structures, having severe consequences

Mass 28 (1), 11

Considerations for route depend on the severity of presentation and patient tolerance to PO medications

In pre-symptomatic PD patients (e.g., those with RBD), this dysbiosis reduces short-chain fatty acid-producing bacteria, further elevating permeability and facilitating CNS infiltration of inflammatory mediators like LPS and -Syn (73)

This process accelerates the proliferation and migration of vascular smooth muscle cells and endothelial cells [38, 39], the HIF signaling pathway under hypoxia/normoxia conditions is shown in Fig