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Interestingly, genetic mouse models of heart-specific CPT I deletion exhibit significant left ventricular systolic dysfunction in response to pressure overload that exacerbates progressively with duration of CPT I gene knockout (He et al., 2012

Taken together, these findings demonstrate that oral butyrate suppresses the mTORC1ISR axis both peripherally and centrally, thereby attenuating PBAT-MPs-induced microglial lipotoxicity, neuroinflammation, and cognitive decline via the gutbrain axis

doi: 10.1038/364626a0 124 LiuY.CaoX.HeC.GuoX.CaiH.AierkenA.et al

Ewha Med J

Neuroinflammation, oxidative stress and the pathogenesis of Alzheimers disease