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Role of the Akt/FoxO3a pathway in TGF-beta1-mediated mesangial cell dysfunction: a novel mechanism related to diabetic kidney disease

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Impaired insulin-stimulated glucose transport (insulin resistance) is thought to be due to reduced GLUT4 delivery to the cell surface, which has been measured in both human muscle and adipose tissue [4,5]

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The loss of the chondrocytic phenotype and proliferative potential during in vitro expansion, termed as dedifferentiation, has been well documented