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Disclosures: Kathryn Schmidt: Nothing to Disclose, Jessica Grimm: Nothing to Disclose, Devin Copley: Nothing to Disclose, Daniel Penrice: Nothing to Disclose, Barbara Copeland: Nothing to Disclose, William Harmsen: Nothing to Disclose, Rachel Gullerud: Nothing to Disclose, Winnie Fan: Nothing to Disclose, Jordan Coffey: Nothing to Disclose, Julianne Lunde: Nothing to Disclose, Sarah Harper: Nothing to Disclose, Hugo Vargas: Sequana Medical: Consultant, Mallinckrodt: Consultant, Ocelot: Grant/Research Support, Arrowhead Phama: Grant/Research Support, Takeda: Grant/Research Support, Durect: Grant/Research Support, River2Renal: Grant/Research Support, Genfit: Grant/Research Support, Kezar Pharmaceuticals: Grant/Research Support, Blanca Lizaola-Mayo: Nothing to Disclose, Douglas Simonetto: Nothing to Disclose, Nothing to Disclose, Nothing to Disclose, Nothing to Disclose 1764 PREVALENCE OF COMPENSATED ADVANCED CHRONIC LIVER DISEASE (CACLD) IN THE UNITED STATES: INSIGHTS FROM NHANES 2017-2020 Ritik Mahaveer Goyal 1 Apoorva Doshi 2 Supriya Maheshwari 3 Imran Qureshi 1 Udita Gupta 3 David Kim 1 Paul Gaglio 1 , 1 Rutgers New Jersey Medical School, 2 Seth GS Medical College and KEM Hospital, Mumbai, 3 University of Alabama at Birmingham Background: Compensated Advanced Chronic Liver disease (cACLD) is described in the Baveno VI consensus which defines it as a continuum of severe fibrosis and cirrhosis as these patients are clinically inseparable

Abstract Parkinsons disease (PD) is one of the most common degenerative brain disorders caused by the loss of dopaminergic neurons in the substantia nigra (SN)

Many of these pathways overlap on key clinical issues like drug resistance, hypoxia adaptation, and CSC maintenance, indicating that targeting miRNASIRT1 interactions or their lncRNA/circRNA regulators could improve therapy or help detect the disease earlier

doi: 10.1016/j.freeradbiomed.2010.12.028

Rowiski, R., Kozakiewicz, M., Kdziora-Kornatowska, K., Hbner-Woniak, E