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Several cytokines like interleukin 1, 6, tumor necrosis factor- and transforming growth factor- induces transcriptional upregulation of both HGF residing in fibroblasts and macrophages and c-MET residing in epithelial cells [30]

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Wyatt, H.J

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(5) In a particular study, researchers explored the theory that the peptide BPC-157 might potentially hasten wound healing compared to a control group

In preclinical studies, it inhibited proliferation of a broad range of tumor cell lines (cell IC50 6.2 nM to 4.3 M) and inhibited HUVEC growth in response to HGF and VEGF as an in vitro model of angiogenesis [116]