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Overexpression of GPx1 in PDA cells in glucose-free medium promotes cell death To further confirm the above results, we tested whether the overexpression of GPx1 could inhibit autophagy in PDA cells upon glucose deprivation

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Research indicates that the IGF-1 signaling pathway enhances mitochondrial antioxidant capacity, reduces ROS generation, and promotes mitochondrial DNA repair by activating the downstream PI3K/Akt pathway ( The mechanism of oxidative damage caused by IR to the organism is shown in Figure 2

Roughly 20% of active patients may improve spontaneously, while 40% stabilize and another 40% progress further with the disease