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Absorption is fundamentally difficult due to its naturally limited bioavailability

As chronic inflammation can lead to increased gut permeability, also known as leaky gut, the interactions between gut health and neurological function become increasingly relevant [1, 10, 11, 13, 18]

The resulting cysteine is unstable at extracellular level, and it is rapidly autoxidizes to cystine

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Interestingly, however, it is unclear if the sir1 phenotype could be fully restored by further inhibiting GCL activity and if the activation of glutathione synthesis in sir1 would exacerbate the growth phenotype