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Arnicolide D not only increases the production of reactive oxygen species (ROS) and decreases mitochondrial membrane potential in BC cells, but it also significantly promotes the expression of PARP-1, enhances the nuclear translocation of apoptosis-inducing factor (AIF), and reduces the level of AIF in mitochondria, thereby inducing the occurrence of apoptosis in a ROS-dependent manner [154]

These pathways are relevant to research on tissue repair mechanisms in muscles, tendons, ligaments, bone, and gastrointestinal tissues [1,6,11]

In: Hall, R.D, editors Plant Cell Culture Protocols

Oleuropein down-regulated IL-1-induced inflammation and oxidative stress in human synovial fibroblast cell line SW982

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