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223 Royer-PokoraB.KunkelL.MonacoA.GoffS.NewburgerP.BaehnerR.et al

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A pathological excess of ROS - as is associated with CYP induction (65), ultraviolet irradiation, xanthine oxidase activity under conditions of ischaemia-reperfusion, and so on - threatens cell viability by jettisoning just those homeostatic mechanisms that physiological oxidative stress secures (21)

Bajaffer A, Mineta K, Magistretti P, Gojobori T

Defects in hemoglobin (Hb), the RBC membrane, and RBC enzymes are the major causes of intrinsic HA, which are commonly referred to as hemoglobinopathy, membranopathy, and enzymopathy, respectively [4,5,6]