The R code for the differential expression analysis is available at All other data supporting the findings of this study are available within the paper and its supplementary material
Diabetes is a disorder in which an individual develops an abnormally high blood sugar level due to inadequate or lack of insulin production by the pancreas or the inability of the body to respond properly to the hormone
Shinmura K, Tang XL, Wang Y, Xuan YT, Liu SQ, Takano H, Bhatnagar A, Bolli R
Hans Neurath (19092002) emphasized that in addition to being a useful tool for protein structure analysis, limited proteolysis (in a form of proteolytic processing) represents a very important biological process leading to the activation of many proteins (including many proteases themselves), which are synthesized as precursors (Neurath and Walsh, 1976
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Schneider 1 , Jan Sperl 7 , Sona Frankova 7 , Marc Bartel 8 , Holger Zimmer 9 , Markus Zorn 9 , Aleksander Krag 5 , Alice Turner 10 , Christian Trautwein 11 and Pavel Strnad 1 , (1)Medical Clinic III, Gastroenterology, Metabolic Diseases and Intensive Care, University Hospital Rwth Aachen, Aachen, Germany, (2)University Hospital Rwth Aachen, (3)Liver Unit, Vall dHebron University Hospital, Vall d`Hebron Research Institute (VHIR), Barcelona, Spain, (4)Clinic for Pneumology, Vall dHebron Hospital, Barcelona, Spain, (5)Department of Gastroenterology and Hepatology, Odense University Hospital, Odense, Denmark, (6)Department of Medicine, Gastroenterology and Hepatology Unit, Sahlgrenska University Hospital, Gothenburg, Sweden, (7)Department of Hepatogastroenterology, Institute for Clinical and Experimental Medicine, Prague, Czech Republic, (8)Institute of Forensic and Traffic Medicine, Heidelberg University Hospital, Heidelberg, Germany, (9)Department of Internal Medicine I and Clinical Chemistry, Heidelberg University Hospital, Heidelberg, Germany, (10)University of Birmingham, Birmingham, United Kingdom, (11)University Hospital, Rwth Aachen Background: Alpha-1 antitrypsin deficiency (AATD) arises from mutations in SERPINA1 and predisposes to liver cirrhosis
