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While the pathophysiological explanation of an elevated B12 for solid cancers is not elucidated, some authors think that B12 elevation could be secondary to the inflammation induced by the antitumor immune response, with plasma haptocorrin release (transcobalamins I and III) by the inflammatory cells [5,6,29]

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The growth hormone secretagogue ipamorelin counteracts glucocorticoid-induced decrease in bone formation of adult rats