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After a stroke, iron tends to accumulate in the damaged area, where it participates in oxidative stress reactions, leading to more cell damage and inflammatory responses (173)

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Additionally, ATOX1 has been shown to function as a new type of copper-dependent transcription factor that mediates copper-induced cell proliferation [32]

Since the observation of the initial activity of the antioxidant enzymes was being investigated, the animals were treated for a relatively short period (3 weeks)