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Their colonization within the human body stems from intricate interactions with multiple facets of host physiology and metabolism, reflecting a finely tuned relationship

Long-term use is not recommended because of the risks associated with prolonged exposure to stimulant medications

The resulting glutamine surplus aggravates glutamine-induced osmotic stress in the cytoplasm, at the same time rendering more glutamine available for (i) its Slc7a6-mediated exchange for extracellular arginine, which increases intra-astrocytic accumulation of reactive nitrogen species (ii) glutamine transport to astrocytic mitochondria, which leads to mitochondrial dysfunction (the Trojan Horse mode of action)