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Programmed necrosis - a new mechanism of steroidogenic luteal cell death and elimination during luteolysis in cows

Future studies should investigate whether p53 ablation in adipocytes, with concomitant reduction in the abundance of AT macrophages, might lead to differences in weight (re-)gain after return to normal or obesogenic diet, to further test the hypothesis that adipocyte p53 is key in establishing an immuno-obesogenic memory of AT that promotes weight regain

Is BPC 157 a steroid

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References Fukamizu et al

MicroRNAs as potential regulators of glutathione peroxidases expression and their role in obesity and related pathologies