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Discussion The evidence presented here reveals novel activities of DBAN at two stages during the cell cycle, at the G1-S transition and later at damaged replication forks (Fig

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S9C), confirming that the loss of phosphorylation was not due to reduced interaction, but rather to the substitution of the target residue itself

Supplementary material The Supplementary Material for this article can be found online at: Abbreviations IS, Ischemic stroke

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