Approximately 1.1 billion compounds were screened via this platform, and multiple candidate compounds with geroprotective properties that demonstrate structural novelty and target specificity were successfully identified
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The mechanistic data cited throughout this article derive from in vitro fibroblast studies and rodent models, which limits generalizability [1]
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Thus, the increased lifespan and delayed disease onset of SOD1 G93A GPX4 mice were not associated with reduction of SOD1 G93A expression and accumulation
Its cardiovascular benefits arise through dose- and tissue-dependent mechanisms: low concentrations predominantly activate AMPK to enhance endothelial function and reduce myocardial injury (Zilov et al., 2019)
