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Description

Cell 61 (2016) 654666

While mutant mice deficient in Dvl1 or Dvl2 alone were normal, double knockouts of both Dvl1 and Dvl2 exhibit PCP defects (Wang et al., 2005), suggesting a functional redundancy between these 2 members

The role of protein conjugation in the disposition of dinitrofluorobenzene

The accompanying DexKey reactors release each active glutathione molecule at the desired point in the gastrointestinal tract, thus achieving optimal absorption