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In addition, the results of EdU incorporation assays suggested that CAF-derived CM differentially slowed down the G1/S transition in A431 cells, and this effect was partially restored when CM was obtained from NAC or raloxifene-treated T205A CAF cultures

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(104) showed that IF improved cognitive dysfunction, prevented A deposition in the brain, and restored AQP4 polarity in the AD mouse model (APP/PS1 double-transgenic mice)