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It should also be noted that the extracellular signaling by IL-1 is inhibited by excessive ROS, but is permitted by ROS-dependent S-glutathionylation, which protects its functionality [94]

Targeted inhibition of mitochondrial iron overload or ROS accumulation effectively inhibited oxidative stress-induced ferroptosis

Shaham-Niv S, Rehak P, Zaguri D, Levin A, Adler-Abramovich L, Vukovi L, et al

This is likely due to the inherent differences in the Australian population of patients diagnosed with HCC compared to the IMBRAVE050 cohort

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